Natural Remedies for Human Papilloma Virus

Cure Hpv for Good

No matter whatever is the age of a person, Hpv can attack anyone anytime. Cure of Hpv has been one of the most costly one and only provided by renowned drug manufactures but now the secret behind this treatment has been revealed in this product. This book allows you to know all the secrets behind a holistic treatment approach which can be followed by you at the bare minimum price that you have ever imagined. It is done without using any drugs or ointments instead natural are ways which can cure Hpv in the shortest possible time than any other drugs available in the market. In most of the cases even after spending thousands of dollars no effects can be seen but no more. This book has been used by many and the response has been phenomenal. A disease which indeed becomes a shame to disclose to others and deadly as Hiv has been most holistically approached and treated in this book. Order now directly from the website to get the best deal. Before getting panic hearing about cervical cancer which is the need of this disease.Order now and save life in a cheapest and natural way.

Cure Hpv for Good Overview


4.6 stars out of 11 votes

Contents: Ebook
Author: Dr. Sylvia Brown
Price: $37.95

Download Now

Genital Warts Eradication System

Easily use the system in the privacy of your own home to once and for all completely annihilate your genial warts forever! Start today with no technical or medical know-how. This system will Obliterate, forever, every single last one of your genital warts secretly, and from the privacy of your own bathroom, bedroom or from anywhere else you decide in your own home! Take you from self-diagnosis all the way to complete and utter freedom from your infection! In short this system is the Ultimate Weapon to help you declare victory over genital warts forever! Instant download e-book manual. Use in secret, in the privacy of your own home. Simple, easy to use 2 step system. Guaranteed to destroy your genital warts in Just 5 Days OR Less! Guaranteed to work, no matter how long you have had the infection or how bad it is!

Genital Warts Eradication System Overview

Contents: EBook
Author: Aston Christiansen
Official Website:

Human Papilloma Virus HPV vaccine

In )une 2006 the US FDA licensed the first vaccine developed to prevent cervical cancer and other diseases in females caused by certain types of genital human papillomavirus (HPV) types (6, 11, 16 and 18), responsible for 70 of cervical cancers and 90 of genital warts. The duration of the protection is unclear. Current studies (with 5-year follow-up) indicate that (he vaccine is effective for at least 5 years (Anonymous 2006), The vaccine has not been causally associated with adverse outcomes of pregnancy or adverse reactions (o the developing fetus. However, data on vaccination in pregnancy are limited.

Human papilloma virus vaccine

Cervical cancer is the second most common cause of cancer deaths in women world-wide. It is almost invariably associated with human papilloma virus (HPV) infection. HPV type 16 is found in about 50 of cervical cancers, and types 18, 31, and 45 account for a further 25-30 of HPV-positive tumors. The development of a safe and effective HPV vaccine could prevent premalignant and malignant disease associated with HPV infection. There has been a trial of a papilloma virus (HPV16) virus-like particle vaccine in 72 healthy volunteers, aged 18-27 years (33C). The vaccine was well tolerated and highly immunogenic. Clinical studies with a recombinant vaccine (using vaccinia virus expressing HPV 16, 18 E6 and E7 proteins) in patients with pre-invasive and invasive cancer have been reviewed (34R).

General Information

In an open study of the use of intralesional cidofovir in treating laryngeal papilloma, 14 adults received monthly injections of cidofovir (maximum dose 37.5 mg per injection in 6 ml of saline mean 22.5 mg) (2). Remission was achieved in all cases with an average of six injections and without additional laryngeal scarring, vocal cord damage, or systemic adverse effects.

Studies reporting deleterious effects on immunity and host resistance

In contrast, other studies have suggested that cannabinoids or marijuana exert deleterious effects as they relate to the immune system and resistance to infection in humans. Gross et al. (1991) indicated that marijuana consumption altered responsiveness of human Papillomavirus (HPV) to systemic recombinant interferon alpha 2a treatment. Tindall et al. (1988) conducted immunoepidemiological studies using univariant and multivariant analyses and implied an association between marijuana use and progression of HIV infection. Caiaffa et al. (1994) indicated that smoking illicit drugs such as marijuana, cocaine, or crack, Pneumocystis carinii pneumonia, and immunosuppression increased risk of bacterial pneumonia in HIV-seropositive users. More recently, Whitfield et al. (1997) examined the impact of ethanol and Marinol marijuana usage on HIV+ AIDS patients undergoing azidothymidine, azidothymidine dideoxycytidine, or dideoxyinosine therapy. In HIV+ AIDS patients with the lowest CD4+ counts...

Implications Of Aberrant Jak Activation In Hematological Diseases

The essential role that JAKs play in normal hematopoietic regulation has been shown by gene-targeting studies that have identified characteristic signaling defects. JAK1 and JAK2 knockout mice are perinatal or embryonic lethal, whereas JAK3 nullizygous mice show severe combined immunodeficiency (SCID) (Table 1) (described in more detail in refs. 9 and 62). Consequently, inappropriate inhibition or absence of JAK activity causes immunosuppressive diseases. The prominent example is the presence of mutations in the JAK3 gene that leads to SCID in humans, characterized by an absence of peripheral T- and natural-killer cells and normal or slightly increased numbers of B-cells (63-66). Other heritable human diseases caused by intrinsic inactivating defects in JAKs have not been reported. However, a variety of pathogens, such as human papilloma virus or cytomegalovirus, are able to inhibit JAK-mediated IFN signaling, thereby escaping immunosurveillance (7,67,68).

Other Than with the Human Immunodeficiency Virus

In contrast, there are reports that cannabinoids and marijuana exert deleterious effects on immune function and host resistance. Juel-Jensen (1972) indicated anec-dotally that individuals infected with HSV who were marijuana smokers had an increased recurrence of genital viral lesions. Also, Harkess et al. (1989) reported on six unrelated outbreaks of hepatitis A among users of marijuana and intravenously administered methamphetamine. Although the exact mode of transmission could not be determined, it was indicated that practices associated with illicit drug use could have facilitated transmission of hepatitis A. Gross et al. (1991) reported that marijuana use altered responsiveness of human papillomavirus to systemic recombinant IFN- 2a treatment and suggested that THC could be a cofactor influencing the outcome of infection. Liauet al. (2002) investigated the association between biologically confirmed marijuana use and laboratory-confirmed sexually transmitted diseases and condom...

Effects Related to Infection with Human Immunodeficiency Virus and AIDS

Difranco et al. (1996), through the San Francisco Men's Health Study (SFMHS), evaluated in a 6-year follow-up study the association of specific recreational drugs and alcohol with laboratory predictors of AIDS. No association with progression to AIDS was observed for marijuana use. Wallace et al. (1998) examined risk factors and outcomes associated with identification of Aspergillus in respiratory specimens from individuals with HIV disease as part of a study to evaluate pulmonary complications of HIV infection. Cigarette and marijuana use was found not to be associated with Aspergillus respiratory infection. Persaud et al. (1999) conducted a cross-sectional survey among 124 street- and brothel-based female commercial sex workers in Georgetown, Guyana, to determine the seroprevalence of HIV infection and describe the sexual practices and drug use patterns. No statistically significant association was found between HIV infection and marijuana use. Miller and Goodridge (2000) evaluated...

Preadministration Assessment

Before administering an estrogen or progestin, the nurse obtains a complete patient health history, including a menstrual history, which includes the menarche (age of onset of first menstruation), menstrual pattern, and any changes in the menstrual pattern (including a menopause history when applicable). In patients prescribed an estrogen (including oral contraceptives), the nurse obtains a history of thrombophlebitis or other vascular disorders, a smoking history, and a history of liver diseases. Blood pressure, pulse, and respiratory rate are taken and recorded. The primary health care provider usually performs a breast and pelvic examination and obtains a Pap smear before starting therapy. He or she may also order hepatic function tests.

Educating the Patient and Family

In most instances, the primary health care provider performs periodic examinations, for example, laboratory tests, a pelvic examination, or a Pap smear. The patient is encouraged to keep all appointments for follow-up evaluation of therapy. The nurse includes several points in a teaching plan.

Reproductive system

There is a positive correlation between high copper loss from an IUCD and the development of menorrhagia or pathological lesions, such as cervical dysplasia and endo-metrial cytopathology (27). Evidence of endometrial carcinoma was not found in endometrial aspirates from 189 women who had used Copper-T-200 devices for 1-10 years, but five cases of endometrial hyperplasia (2.67 ) were encountered in women in the series, all of whom had worn copper devices for 6 years or more. Inflammatory changes in the endometrial cells were found in 12 cases (6.2 ), 11 of 12 having worn the device for over 3 years. It is possible that constant exposure to copper may be responsible for persistence of chronic inflammatory changes in endometrial cells, which could be the precursors of hyperplastic changes. It is not clear whether the dissolved copper is also responsible for the temporarily increased predisposition to bacterial contamination and the somewhat increased risk of pelvic inflammatory disease,...

Ongoing Assessments

At the time of each office or clinic visit, the nurse obtains the blood pressure, pulse, respiratory rate, and weight. The nurse questions the patient regarding any adverse drug effects, as well as the result of drug therapy. For example, if the patient is receiving an estrogen for the symptoms of menopause, the nurse asks her to compare her original symptoms with the symptoms she is currently experiencing, if any. The nurse weighs the patient and reports a steady weight gain or loss. A periodic (usually annual) physical examination is performed by the primary health care provider and may include a pelvic examination, breast examination, Pap smear, and laboratory tests. The patient with a prostatic or breast carcinoma usually requires more frequent evaluations of response to drug therapy.

Antiinfective agents

No risk during pregnancy has been identified for the virostatic acyclovir (see also extensive experience with sytemic use during pregnancy, Chapter 2.6) or for local treatment for condylomata acuminata (venereal warts) with podophyllotoxin, a plant-based mitosis inhibitor (Robert 1994, Bargman 1988, Kami 1980). There are reports on eight pregnant women with condylomata acuminata who were treated with imiquimode, an immune modulator and virostatic for topical application. Two were exposed during the first trimester all newborn were healthy (Einarson 2006, Maw 2004). Recommendation. Anti-infectives may be used on the skin, the mucosa, and even in the eye and ear for the appropriate indications during pregnancy. For theoretical reasons, the best-tested substances should also be the first choice for topical usage however, chloramphenicol should not be used. For usage over wide skin areas, preparations should be viewed as critically as systemic usage because of the danger of absorbing...

Applications in Head and Neck Cancer

Radiotherapy Imrt Head And Neck

Cases of H& N cancer diagnosed in the United States with over 500,000 cases worldwide (American Cancer Society 2005). Tobacco and alcohol use are major risk factors for the development of H& N cancer. For selected H& N tumors, data also implicates Epstein-Barr virus (EBV) and human papilloma viruses (HPV) in the pathogenesis (GiiiiSON et al. 2000). Forastiere AA, Goepfert H, Maor M et al (2003) Concurrent chemotherapy and radiotherapy for organ preservation in advanced laryngeal cancer. N Engl J Med 349 2091-2098 Garden AS, Harris J, Vokes EE et al (2004) Preliminary results of Radiation Therapy Oncology Group 97-03 a randomized phase II trial of concurrent radiation and chemotherapy for advanced squamous cell carcinomas of the head and neck. J Clin Oncol 22 2856-2864 Gillison ML, Koch WM, Capone RB et al (2000) Evidence for a causal association between human papillomavirus and a subset of head and neck cancers. J Natl Cancer Inst 92 709-720

Contraception misconception

The irresponsible actions of some pharmacists and the ludicrous policies of the Bush administration are not the only serious issues in contraception. There are several Republican congressmen, including Senator Coburn from Oklahoma, who happens to have an MD, who have been pressuring the FDA to require a label on condoms that indicate that they do not provide protection against passing or contracting several sexually transmitted diseases, especially Human Papilloma Virus (HPV), which causes genital warts and most cases of cervical cancer. Their efforts are based on a law signed in

Coppercontaining intrauterine contraceptive devices

There is a positive correlation between high copper loss from an intrauterine contraceptive device and the development of menorrhagia or pathological lesions, such as cervical dysplasia and endometrial cytopathol-ogy (14). Evidence of endometrial carcinoma was not found in endometrial aspirates from 189 women who had used Copper-T-200 devices for 1-10 years, but five cases of endometrial hyperplasia (2.67 ) were encountered in women in the series, all of whom had worn

Vein therapeutics and other local therapeutics

Genital or veneral warts are condylomata acuminata caused by the human papilloma virus (HPV) of several types, especially 6 and 11. Treatment is usually by electrocautery, laser, cryotherapy, or surgical excision. Chemical abalation can be used, and involves the use of topical antimitotics such as podophyllotoxin, podophyllin, or 5-fluorouracil, or caustics such as trichloroacetic acid. Interferon-inducers such as imiquimode are also used, and again require multiple applications. Information about these chemicals while breastfeeding is not available, but both the area treated and the dosage are small. Interferon is known to be a large molecule that does not pass into milk even during systemic treatment. The development of the HPV vaccine has shown a decrease in the incidence of these lesions, and will perhaps decrease the need for these medications.

Pharmaceutical potential

Cytotoxic properties Momordica charantia L. has attracted a great deal of interest on account of a series of oleanolic acid saponins known as momordin. Momordin are anticarcinogenic in rodents through the enzymes of biotransformation and detoxification (Ganguly C etal., 2000). Oleanolic acid momordin I, Id and le reduce in vitro the Jun Fos-DNA interaction which is a crucial factor in transmitting tumor-promoting signals from the extracellular environment to nuclear transmission machinery (Lee DK et a ., 1998). Topical application of an extract of peels of the fruits of Momordica charantia L. (100 g animal day) reduces the proliferation of 7, 12-dimethylbenz- a -anthracene-induced skin papilloma in mice (Singh A et a ., 1998). Antimutagen principles characterized from the green fruit are identified as and its stearyl derivative. At a dosage range of 50.5 g extract g-12.5 g extract g in mice, the mixture reduces by about 80 the number of micronucleated polychromatic erythrocytes caused...

Anticancer Effects

Fang et al. reported that rat thioredoxin reductase activity in thioredoxin-dependent disulfide reduction was inhibited by curcumin 50 . By using mass spectrometry and blotting analysis, they showed that this irreversible inhibition by curcumin was caused by alkylation of both residues in the catalytically active site (Cys (496) Sec (497)) of the enzyme. Kang et al. reported that exposure of human hepatoma cells to curcumin led to a significant decrease of histone acetyla-tion 51 . Curcumin can selectively downregulate transcription of human papillomavirus type 18, which is etiologically associated with development of cancer of the uterine cervix in women, as well as activator protein 1 (AP-1) binding activity in HeLa cells. Most interestingly, curcumin can reverse the expression dynamics of c-fos and fra-1 in this tumorigenic cell line 52 .


Dermal treatment with 5-fluorouracil is contraindicated in pregnancy, apart from for the treatment of an individual wart. Treatment of vaginal warts should be postponed until after the birth, or other treatments (such as cryotherapy) used instead. Local use of the cytostatic is, however, not an indication for interruption of pregnancy or for invasive diagnostic procedures.

Pro Apoptotic Agents

Tumor resistance is commonly caused by a loss of the tumor cell's ability to enter apoptosis therefore, modulation of specific molecular pathways leading to increased cell death could widen the therapeutic window. Enhancing apoptotic cell death by modulating the survival pathway and combining this with radiation induced cell killing may be synergistic. This may be more relevant in cervical cancer as the normal apoptotic signaling pathways may be disrupted by the human papilloma virus (HPV) genome (Houcardy et al. 2005). The extrinsic apoptotic pathway is initiated by activation of death receptors on the cell membrane. Apoptosis is triggered by binding of tumor necrosis factor (TNF) receptor super family ligands such as TNF related apoptosis inducing ligand - TRAIL - to their cognate receptors. Normal keratinocytes are relatively resistant